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The ruthenium chloride hydrate (128 mg, 0.57 mmol) was dissolved in ethanol anhydrous (6.2 mL)

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Lung cancer progression is fueled by ROS-induced EGFR mutations and DNA damage, while coronary heart disease arises from endothelial dysfunction and LDL oxidation

Although several pieces of data suggest a possible therapeutic effect of GSH, administered directly or through its precursor N-Acetil Cystein (NAC), in drug-induced liver disease (DILI) [112,113], no randomized-controlled clinical trials are available to support this evidence

In addition, antioxidants that protect the liver from ROS damage and lipid peroxidation may be depleted: GSH, vitamin E, vitamin C and beta-carotene were found to be reduced in the NASH setting (38, 39)