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Ogrodnik et al., 2021), such as dasatinib and quercetin, work by eliminating senescent cells, which reduces the production of pro-inflammatory cytokines including TNF, IL-6, and IL-8, as well as alleviating activation of signaling pathways such as NF-B and JAK/STAT3key contributors to chronic inflammation ( Despite these promising results, questions remain about the duration of benefits and potential adverse effects of long-term senolytic use, which must be addressed through clinical trials before broad implementation

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Finally, epigenetic integration of damage signals, as a hardly neglected cause of stem cell and organismal ageing, has brought new hope for the translational pathway

PK/PD linkage - Controlled human supplementation trials have demonstrated dose-dependent induction of Nrf2 target genes (HO-1, NQO1) in peripheral blood mononuclear cells and reduced selected inflammatory biomarkersproviding mechanistic PD endpoints that can be used to establish pharmacologically active exposure levels for future infectionadjunct trials