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The authors show that the drastic growth arrest of the sir1 mutant is partially rescued when glutathione deficiency is introduced via the cad2 mutation, which decreases glutathione levels to 30% to 40% of wild-type levels (Cobbett et al., 1998), or pharmacologically through treatment with the GCL inhibitor buthionine sulfoximine

In The Molecular Targets and Therapeutic Uses of Curcumin in Health and Disease (pp

[PubMed] [Google Scholar] 59.Jollow D.J., Mitchell J.R., Potter W.Z., Davis D.C., Gillette J.R., Brodie B.B

This synthetic phosphopeptide reached Phase III clinical trials, making it one of the few peptide-based therapies to advance that far in autoimmune disease research

The brain has a high lipid content, which is the main target for oxidative damage