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Clearly, the most successful strategy for targeting GHR signaling to date has involved protein-based therapeutics

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This suggests that a resistance exercise-related mechanism of inducing plasma NO, perhaps due to increased shear stress that triggered an up-regulation in NO-cGMP signaling along with a slow, sustained release of NO from GSH + CIT, may be a conceivable candidate for this response

Lai B, Wu CH, Wu CY, Luo SF, Lai JH