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These results indicated a dynamic polarization plasticity during inflammatory progression, as evidenced by the transition from M1-dominance in acute infection to M2-preference in chronic phases ( In vivo , chronic pulmonary inflammation resulting from prolonged exposure to CS can induce the highly expressed CD206 and TGF- in macrophages, driving the adaptive immune response of the M2 phenotype to participate in tissue repair ( In vivo and in vitro , M2-directed polarization related to the TGF-/Smad pathway in COPD was observed ( As illustrated in Figure 1 , AMs play an important role in the pathogenesis of COPD by driving chronic inflammation and tissue destruction

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RPG group exhibited decreased relative abundance of pangamic acid ( P = 0.033), 6-hydroxypentadecanedioic acid ( P = 0.038), 5,6-dihydroxytetradecanedioic acid ( P = 0.005), 3-(4-hydroxyphenyl)-lactate ( P = 0.045), 5-hydroxyindole-3-acetic acid ( P = 0.006), (2-oxo-2,3-dihydro-1H-indol-3-yl)-acetic acid ( P = 0.038), and glutamic acid ( P = 0.038) but increased alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid versus RUG group

doi: 10.1007/s00726-009-0377-x 167