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AngQYAlbaDLUpadhyayVBisanzJECaiJLeeHLet al

2024 ASCO Annual Meeting 42(17), 2024

These therapeutic strategies possess high selectivity, especially when activated by GSH localized in the tumor [80]

Several clinical studies have found that low expression levels of AREG and EREG are related to the low response rate to cetuximab in KRAS-WT CRC patients, but not in CRC patients with KRAS-mutation ( 4.1 Strategies to overcome resistance caused by mutations in EGFR in mCRC A second-generation FDA-approved human EGFR mAb, necitumumab has been shown to bind to EGFR that harbors all reported cetuximab- and panitumumab-resistant mutations except for those at position G441 ( Oligoclonal combination inhibitors that can bind to the mutated ECD of EGFR are new approaches to targeting EGFR that are expected to overcome resistance caused by EGFR ECD mutations