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The expression of the endogenous mTORC1 inhibitor sestrin 2 is induced by UVB and balanced with the expression level of sestrin 1

This bioenergetic and oxidative failure is followed by defective organelle quality control, particularly insufficient mitophagy, autophagic blockade, lysosomal dysfunction, and ineffective clearance of damaged cellular components

Blockade of interleukin 6 signalling ameliorates systemic insulin resistance through upregulation of glucose uptake in skeletal muscle and improves hepatic steatosis in high-fat diet fed mice