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For example, ROS can activate nuclear factor kappa B (NF-B), a key transcription factor that regulates the expression of pro-inflammatory cytokines, chemokines, and adhesion molecules [134]

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76 In a cell experiment, when renal tubular cells experienced hypoxia and then reoxygenation, the levels of HIF-1 and BNIP3 were increased, mediating mitophagy, which could protect cells from cell apoptosis

This neuroinflammatory response is associated with cognitive impairment, upregulation of tau and reduced synaptopodin in zebrafish [81]