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They also included the coupling helices ICL5 and ICL7 and their associated TMs 10-11 and 14-15, respectively, due to their role in substrate transport 34,35

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Recently, a correlation between KRT82 gene mutations and increased susceptibility to AA has been identified, proposing that dysfunction of keratin might compromise the keratinizing layer of the hair follicle in these patients (Erjavec et al