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Abstract Acquired methemoglobinemia is a common, potentially fatal syndrome that can occur as a result of exposure to numerous xenobiotics

However, a key limitation remains in achieving efficient and uniform CRISPR/Cas9-based transduction within intact 3D organoid structures

All studies written in preclinical and clinical language that highlighted the anticancer molecular mechanisms, signaling pathways and action targets of bromelain were included

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This effect is achieved by increasing HO-1 activity and signaling to biliverdin reductase (BVR)