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Research demonstrates that BPC-157 upregulates vascular endothelial growth factor receptor-2 (VEGFR2) at both mRNA and protein levels in endothelial cells without increasing VEGF-A itself, instead sensitizing cells to existing VEGF signaling while simultaneously activating the Src-Caveolin-1-endothelial nitric oxide synthase (eNOS) pathway

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For comparison: BPC-157 modulates hundreds of genes TB-500 hundreds to thousands GHK-Cu modulates 31.2 % of all human genes , an exceptionally broad pleiotropic profile This is why GHK-Cu is called the master regulator in the dermatological and regenerative research literature