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INTRODUCTION Acute liver failure consists of severe liver dysfunction, as evidenced by coagulopathy, jaundice, and encephalopathy, usually in the absence of underlying liver disease[1]

By modulating immune responses characterized by reduced levels of pro-inflammatory markers (TNF-, IFN-) combined with increased Th2 and Treg cell ratios and TGF-1 concentrations

3 vCD44BP constrains effector but not memory CD8 T cell numbers

A comprehensive focus on both safety and functionality is critical for the selection of next-generation probiotics

doi: 10.1007/s12011-016-0768-2 93 StojsavljeviARovaninBJagodiJKrstiPaunoviIGavrovi-JankuloviMet al