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It does not indicate a problem with the peptide

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Furthermore, miR-483-5p was shown to exacerbate cisplatin-induced AKI by targeting GPX3, whereas viral-mediated overexpression of GPX3 prevented AKI by reducing oxidative stress and apoptosis in tubular cells [41]

The nitric oxide generated through this pathway promotes vasodilation, inhibits platelet aggregation, and enhances endothelial cell survival while supporting the formation of patent, functional vessels

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