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There are counteracted muscle wasting, significantly corrected deranged muscle proliferation as well as myogenesis, counteracted increase of the proinflammatory cytokines such as IL-6 and TNF-, looking at muscle metabolism relevant to cancer cachexia as well as the changes in the expression of FoxO3a, p-AKT, p-mTOR, and P-GSK-3 (Kang et al., 2018)

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These results together confirm that the c-MET II-HGF I interface as observed in the structure is important for the HGF-induced activation of c-MET

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