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As previously demonstrated, glpD / golD / uhpT mutants display a 12 log defect in both the spleen and liver, while ptsI mutants were 500-fold and 5000-fold attenuated in the spleens and livers of infected mice respectively (Fig 5A)

I genuinely wanted to understand what their reasoning was so I could present a balanced view and write a paragraph or two about it in a blog post to raise awareness and help others advocate for themselves

E.JenkinsN

[1] The absence of any signal in the published literature is consistent with the mechanistic analysis above showing no shared metabolic pathway

The data for the six-month period from October 2022 to April 2023 was juxtaposed with the corresponding period in the prior year