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A., Renal Dysfunction Might be a Marker of Cardiovascular Dysautonomia in Prodromal synucleinopathies, Movement Disorders 35, no

PKC pathway in diabetic cardiovascular complications The hyperglycemic activation of PKC may serve as an associated mechanism through which oxidative stress and AGEs exert their detrimental effects on CVS [44], with its activation further stimulated by oxidative stress, angiotensin II, platelet-derived growth factor (PDGF), and vascular endothelial growth factor (VEGF), collectively intensifying its pathological impact

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The consistency of findings across sensitivity analyses underscores the robustness of the effect size estimation

Lastly, the catabolic machinery, including monoamine oxidase A and B ( MAOA , MAOB ) and catechol- O -methyltransferase ( COMT ), is expressed in human and mouse -cells and -cells (Fig