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These results confirm that ubiquitylation of IL-1 at K133 is required for efficient proteasomal degradation of TLR-induced IL-1

Ascites is a buildup of fluid in the belly, often due to severe liver disease

Iron interacts with -synuclein, promoting the formation of Lewy bodies (50), which are hallmark features of synucleinopathies such as Parkinsons disease, Lewy body dementia, and multiple system atrophy (51)