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Given these functions, GlyT1 has been explored as a therapeutic target for drug development, particularly in psychiatric and neurological disorders

, Kim1 expression increased with higher doses of sulfasalazine (Supplemental Figure 6), suggesting a dose-dependent nephrotoxic effect

[3] Langova, L., Novotna, I., Nemcova, P., Machacek, M., Havlicek, Z., Zemanova, M., Chrast, V