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Naringin could accelerate mitochondria-mediated cells apoptosis by generating mitochondrial membrane potential depolarization, by increasing the expression level of Bax, by releasing Cyt c and activating caspase-3 in VK2/E6E7 and End1/E6E7 cells

Abstract Hepatic ischemia/reperfusion (I/R) injury, caused by limited blood supply and subsequent blood supply, is a causative factor resulting in morbidity and mortality during liver transplantation and liver resection

2013;8: e60115

Recently, catalytic anticancer architectures that target biochemical traits unique to tumor microenvironment have aroused multidiscipline interest

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