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[Google Scholar] Selkoe, D

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During TB, IFN-I signaling-mediated recruitment of inflammatory monocytes augments the intracellular permissibility of monocytes and promotes their differentiation into an Mtb-tolerant M2 macrophage cell type with a non-inflammatory phenotype, while reducing monocyte differentiation into dendritic cells and impairing antigen presentation and T-cell priming (142)