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Description

This study provides important insights into the molecular mechanisms of ferroptosis in RA and the development of new therapeutic strategies

J., Patabendige A

& Kato, J

Only the lack of OppC and GsiC in GSH-deficient cells ( gshA gsiC oppC ) completely inhibited GSH-depended reduction of Grx1-roGFP2, whereas the other triple mutants were unaffected (Figure 4B-E)