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During the pathogenesis of OA, JUN directly binds to the promoter of NCOA4 and initializes its transcription, increasing ferritinophagy degradation, leading to ferroptosis and extracellular matrix degradation in chondrocytes

Building on previous findings, the current research aimed to assess the protective roles of BCA, CoQ10, and PHL against oxidative stress, nephritic apoptosis, and molecular disruptions caused by simultaneous chromium and arsenic exposure in Swiss albino mice

Obstructive sleep apnea and comorbidities: a dangerous liaison

Newer combinations of drugs and multimodal treatment regimensimplemented in both neoadjuvant and adjuvant settingshave shown promise in extending survival

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