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The defense mechanism indicates that the number of ROS the human body produces can be managed without causing damage

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These findings suggest that pathological cardiac pressure overload induces NNT reverse functional mode to generate NADH at the expense of NADPH resulting in a high NADH/NAD + and reductive stress, further promoting ROS at complex I leading to oxidative injury and cell death (Fig

Nat Rev Cancer 3 (9): 639-49, 2003