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Myeloid cells (i.e., monocytes, macrophages, neutrophils, and dendritic cells) have been believed to be effector cells that drive the initiation of inflammation, and dysfunctions in the immunoregulatory roles of these cells may play a part in the pathogenesis of IBD [16]

Additionally, the extract fractions did not show relevant cytotoxicity against kidney (BGM) and liver (HEP2) cell lines, indicating a selective safety margin for the active compounds (Frana et al., 2014)

Specifically, neutrophils are provided by two different mechanisms: The first one is called paracellular transmigration and consists of a connection with the apical domain of ECs via cell surface proteins (e.g., pseudopods) over one or between two ECs