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BMP activation drives hepatic bias in GPX2-deficient PFG To elucidate the molecular basis of the PFG differentiation bias observed in KO cells, we leveraged RNA-seq and scRNA-seq datasets to analyze differential signaling pathway activity across key developmental stages: PFG, PEN, and SC- cell

All this evidence demonstrates the therapeutic potential of FAK inhibition

10.3390/ijms24097910 Pubmed Abstract CrossRef Google Scholar View reference in article 33

This is practical rather than strictly necessary for tolerance management

A False-Positive Screening Hit in Fragment-Based Lead Discovery: Watch out for the Red Herring