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More direct evidence for the translocation of zinc was provided by the findings of Koh and others (11), who demonstrated that during a brief period of global ischemia, intracellular zinc accumulation in vulnerable CA1 pyramidal hippocampal neurons preceded degeneration, which could be prevented with the intracerebroventricular administration of the high affinity, membrane-impermeable zinc-chelator, ethylenediaminetetraacetic acid (EDTA) saturated with calcium (Ca-EDTA)

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Tomita K, Smith PM, Takayama M, Pan C, Shen S, Wang X, Wang XS, Williams LA, Arvide EM, To C, Rajaram R, Mitchell KG, Rice DC, Hofstetter W, Antonoff MB , Mehran RJ, Vaporciyan A, Walsh GL, Maxwell JE, Snyder RA, Kim MP, Tzeng CW, Mansfield PF, Swisher SS, Lee J, Badgwell BD, Katz M, Ikoma N

doi: 10.1136/jitc-2020-001372 17 ZouZChangHLiHWangS

Yu X, Li Y, Zhang Y, Yin K, Chen X, Zhu X