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The biggest obstacle to the discovery of therapeutics for sepsis is its diverse etiology among patients (55), which have prompted researchers to study the molecular mechanism of sepsis progression based on systemic inflammatory responses

doi: 10.1038/ncb3272 153 BottAJPengICFanYFaubertBZhaoLLiJet al

Subsequently, respiratory infection of F2 hybrids (DBA/2 x C57Bl/6) with Mtb H37Rv revealed a major locus on chromosome 19 ( Trl-4 ) to be involved in bacterial replication in the lungs (Mitsos et al., 2003)