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Therefore, DHODH/CoQH 2 and mitochondrial GPX4 constitute two major ferroptosis defense systems to eliminate cell damage or death caused by the accumulation of mitochondrial lipid peroxides, and preclinical studies have shown that targeting DHODH can promote ferroptosis[36]

Agents such as metformin, rapamycin, or trehalose can rejuvenate autophagy and mitophagy, thereby clearing damaged mitochondria and lipid droplets to reduce mitochondrial ROS and inflammatory priming

Medesan C, Cianga P, Mummert M, Stanescu D, Ghetie V, Ward ES

Chan SSM, Luben R, et al

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