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Liver-specific deletion of acetyl-CoA carboxylase 1 reduces hepatic triglyceride accumulation without affecting glucose homeostasis

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The gastrointestinal side effects at higher doses are too significant for most people to tolerate without a gradual buildup period

A mismatch can develop between the haptocorrin-bound pool, which makes up most of the total, and the smaller active fraction on transcobalamin that cells depend on

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