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In p53-induced ferroptosis, the p53 protein activates arachidonate-12-lipoxygenase (ALOX12), thus inducing cell death via ferroptosis, independent of GPX4 activity [46]

Conversely, the clearance of mtROS using MitoQ or the genetic deletion of NOX2 results in reduced IFN- production and inhibited Th1 differentiation, suggesting that ROS from different sources positively promotes Th1 differentiation in IBD