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The esr1-1 (enhanced stress response 1) mutant was identified conferring enhanced resistance to the fungal pathogen F

Protective role for b-1b b cells and IgM in obesity-associated inflammation, glucose intolerance, and insulin resistance

Biomechanically, the healed tendons (over 14 days) showed an increased load to tendon failure and significantly higher functionality (Achilles functional IndexAFI)

For estrogen pathways, GHKCu appears to influence aromatase activity in some models, with potential to lower local estrogen production where excess drives pathology

Structural basis for Epstein-Barr virus host cell tropism mediated by gp42 and gHgL entry glycoproteins