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Concurrently, alterations in lipid metabolism indicate the necessity for membrane remodeling and energy reallocation during salinity stress, both of which are crucial for sustaining homeostasis and physiological functions in dynamic environments

Solomon, I

These therapeutic ambitions are further tempered by the practical difficulties of achieving cell-type-specific modulation without disrupting homeostasis or compromising delivery across the bloodbrain barrier

These additions strengthen mechanistic links between intestinal dysfunction and hepatic pathology

It is especially critical in Phase II liver detox pathways