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Additionally, the endogenous administration of BDNF was shown to protect neurons within the substantia nigra from 6-OHDA and MPTP toxicity, both in rat and primate Parkinson models [31]

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This outcome was attributed to MOTS-c restoring metabolic balance in muscle tissue (largely via AMPK activation), underscoring its potential for combating age-dependent metabolic decline

moreover, for five of the 10 antibodies, bioavailability data were only available for the SC formulation with hyaluronidase, with no comparative data for formulations without it, making comparison in these antibodies impossible