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One final convergence point of these related signaling events, is the initiation of the p38 MAPK and MAPK/ERK1/2 signaling pathways, that causes changes in gene expression and can modify cell metabolism and proliferation ( Future GH-IGF Mechanisms to Explore and Concluding Remarks Since steroid hormones are not stored in large quantities in steroidogenic tissues, there is constant demand for cholesterol, the main precursor of steroidogenesis ( in vivo through an unknown mechanism, but the effects of ATGL in steroidogenic cells in the ovary and testes requires further research ( The downstream signaling from FSH-FSHR interactions is clearly central to the life and death balance observed in granulosa cells and developing follicles, and seems to have parallels in male reproductive cells

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The metabolic coupling for taurine synthesis and degradation between astrocytes and neurons, evidence neuronal dependence on astrocytes for the adequate functioning of the mentioned effects in the brain