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The upregulation of long-chain acyl-CoA synthetase family member 4 (ACSL4), an enzyme involved in lipid metabolism and necessary for lipid peroxidation, promotes ferroptosis [25]

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Osteoclast production is thus decreased, as is the expression of osteoclast marker genes triggered by RANKL (125)

Known for its high selectivity, it has minimal impact on cortisol and prolactin levels, making it one of the more refined GH-releasing peptides studied in controlled research settings