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[DOI] [PMC free article] [PubMed] [Google Scholar] 75.Abad C., Gomariz R.P., Waschek J.A

Nat Commun 12(1):7081, 2021

& Francis, F

Nevertheless, thanks to FRDA iPSC-based models, the reasons behind GAA repeat expansion and mechanisms of disease-relevant phenotypes were studied thoroughly and efficient therapies using CRISPR/Cas9 to increase FXN expression were investigated over the past decades

Although none of these cysteines is in the active site, the probability of the modification of one of the cysteines by juglone leading to its inactivation, as seen in the case of Pin-1, Sortase, and RNase P enzymes, seemed a probable mechanism of inhibition