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Copper induces increased levels of inflammation in HepG2 ATP7B-KO cells To assess inflammatory responses, the expression of FOS , PLA2G2A , JUN , and JUNB was analysed in copper-treated HepG2 and HepG2 ATP7B-KO cells

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A vital contribution of the TSS pathway in sustaining cysteine levels is detected only under hemin exposure or physiological but not supraphysiological cystine supplement