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De-lactylation at K412 of HDAC1 activates the transcription of the m6A erasers, fat mass and obesity-associated protein (FTO) and AlkB homolog 5 (ALKBH5), suppressing m6A modification on ferroptosis suppressor protein 1 (FSP1) mRNA and accelerating its degradation, thereby enhancing ferroptosis sensitivity (56)

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The n-6 PUFA biosynthesis pathway and ferroptosis Cells can directly take up AA (C20:4n-6) from their environment, and linolenic acid (LA, C18:2n-6), a precursor of AA and an omega-6 essential fatty acid, is much more abundant in human blood, implying that cells may need to synthesize AA directly from LA (Fig