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TMAO has been implicated in angiotensin II-mediated vasoconstriction (through enhancement of intracellular calcium release), increased sympathetic activation and oxidative stress, and impairment of endothelial nitric oxide production (involved in vasodilation) (Fig

Nature 586 , 434439 (2020)

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[Abstract]2024 Mar 15:328:121689

Neuroprotective effects of benfotiamine Neuroprotective effects of benfotiamine were first observed when it was used in the treatment of neuropathies associated with diabetes [[65], [66], [67]]