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Acknowledgments AS is grateful to Institute of Excellence, BHU for financial support and facilities like computers, devices and peripherals

Matsumoto et al

In the gastrointestinal tract, BPC-157 demonstrates gastric cytoprotection in animal models, reducing ulcer formation and accelerating mucosal healing at doses consistent with tendon repair studies

Despite their proliferative arrest, senescent cells exhibit a metabolically active state characterized by an altered glycolytic flux, perturbed lipid homeostasis, mitochondrial dysfunction, and a disrupted nicotinamide adenine dinucleotide (NAD +) redox balance, all of which contribute to vascular damage