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This review integrates preclinical research related to the effects of both stress and antidepressant treatment on synaptic plasticity, and proposes normalization of synaptic function as a final common pathway for depression treatment

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Gene network analyses identify co-regulated transcription factors and BACH1 as a key driver in rheumatoid arthritis fibroblast-like synoviocytes

In fact, Madin-Darby canine kidney cells or human small airway epithelial cells infected with influenza virus and treated with reduced GSH exhibited an inhibition of viral matrix proteins