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2b)and the emerging recognition of BCL-2 as a survival factor driving castration resistance, here we sought to define how AR +/- BCL-2 +/- PCa cell subtypes dynamically evolve in response to castration and during progression to CRPC, understand how AR signaling controls BCL-2 expression in different androgen signaling environments, and, critically, determine whether BCL-2 represents a shared, therapeutically actionable resistance mechanism across distinct CRPC subtypes

doi: 10.1080/14017430601049449

In case of a therapeutically necessary high dosage, the additional intake of this monoproduct helps avoids the accumulation of the vitamins and trace elements contained in the basic product

Recombinant tissue plasminogen activator protects neurons after intracerebral hemorrhage through activating the PI3K/AKT/mTOR pathway

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