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doi: 10.3390/jof10060394

In cancers where B-Raf is mutated, such as the BRAF V600E mutation, the kinase is constitutively active, resulting in continuous signaling through the MAPK pathway, contributing to tumorigenesis (Bharti et al., 2025[8]

Evidence to support direct effects of H1 receptor antagonism on development of NAFLD is scarce however, H1R antagonism was reported to exacerbate high fat diet induced NAFLD in wild-type mice (Raveendran et al., 2014), suggesting a role of this receptor subtype in the pathogenesis of NAFLD

Unloaded dyes and organic solvent were removed through dialysis against deionized water for 48 h

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