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Optimal antioxidant levels in the body have been associated with cognitive preservation, and several studies have demonstrated the neuroprotective effects of catechins, highlighting their potential as adjunctive therapy in select neurodegenerative diseases

Altered connectivity in depression: GABA and glutamate neurotransmitter deficits and reversal by novel treatments

(2021) suggested that the absence of the Fas-activated serine/threonine kinase (FASTK) gene may inhibit the ethanol-induced phosphorylation of human antigen R (HuR) and dissociation of the HuR-sirtuin 1 mRNA complex

Our findings raise the possibility that IF-modulated content expulsion under neuronal stress conditions might be an unexpected contributor to disease pathology in these disorders

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