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Cardiac cells that have been genetically engineered to lack NRF2 have been shown to be unable to resist damage due to reactive oxygen radicals, presumably due to a deficiency of SOD (3)

The administration of PET-NaPs resulted in oxidative stress in the brain tissue, as evidenced by a substantial reduction in GSH levels and an increase in MDA levels compared to the control group ( P 0.05)

Sweeps at the voltage-sensitive sodium channel ( VSSC ) gene on chromosome 3 correspond to one resistance-associated nucleotide substitution in Ae

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