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The secondary lesions of AS include lipid deposition, intimal thickening, thrombosis, inflammatory cell infiltration, subintimal inflammation, vessel wall remodeling, neovascularization, plaque rupture, intraplaque hemorrhage, plaque rupture and local thrombosis, which eventually cause narrowing or blockage of the vascular lesion, resulting in ischemic injury to the affected organs (4)

Therefore, we refer to them as putative novel loci that occur adjacent to previously identified association signals and require replication to determine whether they represent independent effects or extensions of known loci

The body produces them when fatty acids are broken down (from fat) as energy sources

nagelii and yeast strains involving Z

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