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Collectively, these findings indicate that non-selective or excessive chelation of brain iron may disrupt physiological metallostasis, emphasizing the need for precisely titrated, brain-targeted anti-ferroptotic interventions with balanced efficacytoxicity profiles. Beyond traditional chelators, several natural compounds have also shown potential in regulating iron transport proteins

Taken together, our data suggest that the TLR-activated PI3K/AKT axis phosphorylates EPRS1 at Ser999 and converts it to an anti-inflammatory immune regulator via association with AKT and, potentially, GSK3 (Fig

FEBS Lett 566:15 Mittler R, Vanderauwera S, Gollery M, Van Breusegem F (2004) Reactive oxygen gene network of plants

healthy repair is organized, appropriately cross-linked, and self-limiting