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Inflammation and oxidative stress are the core pathological features of viral pneumonia: after viral invasion, on the one hand, it activates immune cells (such as macrophages and neutrophils) to release pro-inflammatory factors, such as interleukin-6(IL-6) and tumor necrosis factor-alpha (TNF-), triggering a cytokine storm, leading to the destruction of the alveolar-capillary barrier and pulmonary edema

Flow cytometric analysis corroborated this finding, demonstrating a significant increase in the frequency of LARS2-high cells among moderate-mutant tumor cells (Fig

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Methods: Patients within the Childhood Liver Disease Research Network (ChiLDReN) enrolled in the Longitudinal Study of Genetic Causes of Intrahepatic Cholestasis (LOGIC) study were identified with BASD by local centers via biochemical evidence based on mass spectrometry analysis of urine/serum or from biallelic pathologic mutations in genes encoding enzymes in the bile acid synthetic pathway