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Mutations in neuronal synaptic proteins include post-synaptic proteins such as neuroligins (NLGNs) and pre-synaptic proteins such as neurexins (NRXN), SHANK, and GRIP1 [73]

7a), with the hydroxypyruvate level in glyr1-1 cat2-1 returning to wild-type levels and is 22% of that in cat2 (Supplementary Fig

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By enabling the optimization of treatment regimens, this model may serve as a valuable tool for improving drug safety and therapeutic outcomes