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Study secondary end points included the comparison of ACE1 and ACE2 receptor levels between the supplement and placebo groups of Cohort 2 (analyzed at baseline and every 7 days for 21 days) and the ACE1/ACE2 ratios in both supplement and placebo groups of each cohort

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For example, C16-carnitine (10 M) may impair mitochondrial function in rat ventricular myocytes by accelerating ROS production and increasing the probability of mitochondrial permeability transition pore (mPTP) opening, whereas low concentrations (1 and 5 M) of C16-carnitine lightly hyperpolarize the m but do not open the mPTP [293]

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At the studys end, both the ALCAR group and placebo group worsened in their mental performance testingbut the ALCAR groups mental decline was slower than the placebo groups in 13 of the 14 brain function measures